Preliminary experiments reveal conolidine may inhibit precise ion channels, lowering neuronal excitability and limiting agony signals. This mechanism is particularly related in neuropathic ache, where irregular signaling triggers persistent distress. Moreover, conolidine appears to impact G protein-coupled receptor (GPCR) pathways integral to agony notion. Computational docking resear... https://mohamedz692zuo9.wikipublicity.com/user